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3 edition of Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line found in the catalog.

Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line

Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line

sizing and mapping of virus-specific RNA

by

  • 106 Want to read
  • 17 Currently reading

Published .
Written in English


Edition Notes

Statementby Vicky Lin Christy Van Santen.
Classifications
LC ClassificationsMicrofilm 83/402 (Q)
The Physical Object
FormatMicroform
Paginationv, 113 leaves
Number of Pages113
ID Numbers
Open LibraryOL3205112M
LC Control Number83110393


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Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line Download PDF EPUB FB2

Kieff E. et al. () Persistence and Expression of the Epstein-Barr Virus Genome in Latent Infection and Growth Transformation of Lymphocytes. In: Levine P.H., Ablashi D.V., Pearson G.R., Kottaridis S.D. (eds) Epstein-Barr Virus and Associated Diseases. Developments in Medical Virology, vol 1.

Springer, Boston, MACited by: 7. To detect EBV expression, paraffin sections from 17 LCH cases were examined by mRNA in situ hybridization for EBV BamHIW, Epstein-Barr virus nuclear antigen-2. Epstein-Barr virus (EBV) persists for the life of the host by accessing the long-lived memory B cell pool.

It has been proposed that EBV uses different combinations of viral proteins, known as latency types, to drive infected B cells to make the transition from resting B cells to memory cells. Epstein Barr Virus Latent Membrane Protein Cell Surface Marker Expression Cell Line Establishment Primary Epstein Barr Virus Infection These keywords were added by machine and not by the authors.

This process is experimental and the keywords may be updated as the learning algorithm : Martin Rowe. Vicky Lin Christy Van Santen has written: 'Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line' Asked in Chickenpox What virus is similar to Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line book.

Vicky Lin Christy Van Santen has written: 'Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line' Asked in. The herpesviruses Epstein-Barr virus (EBV) and human herpesvirus 8 and the retrovirus human T-cell leukemia virus type 1 are directly implicated.

@article{osti_, title = {Epstein–Barr virus glycoprotein gM can interact with the cellular protein p32 and knockdown Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line book p32 impairs virus}, author = {Changotra, Harish and Turk, Susan M.

and Artigues, Antonio and Thakur, Nagendra and Gore, Mindy and Muggeridge, Martin I. and Hutt-Fletcher, Lindsey M., E-mail: [email protected]}, abstractNote = {The Epstein–Barr virus. Epstein-Barr virus (EBV; also known as human herpesvirus 4 [HHV-4]) and cytomegalovirus (CMV; also known as HHV-5) logically belong in the same chapter.

Both of these human herpesviruses are major pathogens for the immunocompromised host, and both are capable of causing disease during primary infection, reactivation, or superinfection. They, along with BK Author: Henry H.

Balfour, Kristin A. Hogquist, Priya S. Verghese. Author Summary The Epstein-Barr nuclear protein EBNA3C is a well-characterised repressor of host gene expression in B cells growth-transformed by EBV. It is also well established that EBNA3C can interact with the cellular factor RBPJ, a DNA-binding factor in the Notch signalling pathway conserved from worms to humans.

However, prior to this study, little was known Cited by: We have studied the influence Expression of Epstein-Barr virus genes in an Epstein-Barr virus growth-transformed cell line book BLIMP1β expression of the Epstein-Barr virus (EBV), a human herpesvirus that is implicated in the pathogenesis of several GC-derived lymphomas, including a subset of DLBCL and Hodgkin’s lymphoma (HL).

We show that BLIMP1β expression is increased following the EBV infection of normal human tonsillar GC B by: 7. The Epstein-Barr virus (EBV) is a ubiquitous gamma herpesvirus that was originally identified in Burkitt’s lymphoma (BL) by Tony Epstein and his colleagues in [].It is also the first virus known to play an essential role in the induction of human malignancies [].Interestingly, EBV infection is common among most population groups by: The Epstein-Barr nuclear protein EBNA3C is a well-characterised repressor of host gene expression in B cells growth-transformed by EBV.

It is also well established that EBNA3C can interact with the cellular factor RBPJ, a DNA-binding factor in the Notch signalling pathway conserved from worms to by: Beyond the Provirus: from Howard Temin's Insights on Rous Sarcoma Virus to the Study of Epstein-Barr Virus, the Prototypic HumanTumor Virus, p In Cooper G, Temin R, Sugden B (ed), The DNA Provirus.

ASM Press, Washington, DC. doi: /ch12Author: Bill Sugden. What is claimed is: 1. A viral vector, comprising: an Epstein-Barr virus (EBV) genome, wherein a heterologous nucleotide sequence operatively linked to a promoter is inserted into the major internal repeat region of said EBV genome, and wherein said vector can target B-lymphocyte cells and express said heterologous nucleotide sequence therein.

Epstein-Barr virus expresses a cytoplasmic and plasma membrane protein (LMP) in latently infected growth transformed lymphocytes.

The gene specifying LMP has now been expressed in NIH3T3 and Rat-1 cells. A single male child with severe microcephaly, pre- and postnatal growth retardation, and abnormal skin pigmentation was found to have mitomycin C (MMC)-induced chromosomal breakage in fresh T-lymphocyte cultures, as well as in Epstein–Barr virus (EBV)-immortalized B lymphoblasts ().Striking centromeric cohesion (“railroading”) and premature chromatid.

Establishment of Epstein-Barr virus growth-transformed lymphoblastoid cell lines Hui-Yuen, J., McAllister, S e Research output: Contribution to journal › Article. Transformed Cell Line. Viruses. Human Herpesvirus 4. Non-integrating lentiviral vectors for specific killing of Epstein-Barr virus nuclear antigen 1-positive B cell.

Epstein–Barr virus growth-transformed cells are converted to malignancy following transfection of a kb CATR1 antisense construct independent of a change in the level of c-myc expression followed by a 8;14 chromosomal translocation.

Following infection by a sarcoma virus and intergration of the provirus into the host genome, the overexpression of the incorporated oncogene, or expression of a mutated oncogene that continuously induces the cell to multiply, results in transformation of the infected cell. The incorporation of cellular oncogenes into a retrovirus is an.

Full text of "Report of program activities: National Cancer Institute" See other formats. Epstein--Barr Virus Growth-Transformed Cells are Converted to Malignancy Following Transfection of a Kb CATR1 Antisense Construct Independent of a Change in the Level of c-myc Expression Followed by a 8;14 Chromosomal Translocation (pp.

[Epstein-Barr virus is the known cause of nasopharyngeal cancer. -cast] Li - Int J Cancer abstract / PubMed Chemicals vs HPV in bladder cancer: Papillomaviruses underlie supposed chemical carcinogenesis. With state-of-the-art techniques, human papillomaviruses are now found in virtually % of cervical cancers.

Full text of "Molecular Biology Of Cancer Mechanisms, Third Edition Lauren Pecorino" See other formats. Fig. The virus replication cycle. Most generally, virus replication can be broken into the stages shown: (a) initial recognition between virus and cell and introduction of viral genetic material into the host cell; (b) virus gene expression and induction of virus-induced modification of host allowing; (c) virus genome replication.

The bfl-1 gene is transcriptionally upregulated by the Epstein–Barr virus LMP1, and its expression promotes the survival of a Burkitt’s lymphoma cell line.

J Virol ; – Kawanishi M. Expression of Epstein–Barr virus latent membrane protein 1 protects Jurkat T-cells from apoptosis induced by serum deprivation. Though it is understood that T-cells are a critical component of the immune system’s ability to destroy foreign invaders and attack cancerous cells, very little is known regarding the specific epitopes that are recognized by T-cells to carry out these functions.

Generally, the epitopes mediating this immunity are short, contiguous peptides derived from antigenic proteins Cited by: 1. Calcium phosphate transfection of cells, as a means of obtaining high level expression of genes such as the EBERs, is a useful technique, given the absence of EBV genes in this cell line.

cells are a human embryonic kidney line transformed by the introduction of sheared adenovirus DNA (9).

Aantekeningen H1t/m10 Boek + HC's. Samenvatting en uitwerking H1t/m10 (Boek of HC's) Universiteit / hogeschool. Vrije Universiteit Amsterdam. Vak. Oncologie AB_ Geüpload door. Abel Ferrari. Academisch jaar. 17/ When reproducing, viruses use at least part of the cell’s equipment for replication of viral nucleic acids and expression of viral genes.

They also use the cell’s protein synthetic machinery, and the cell’s metabolic energy resources. The dimensions and organization of “typical” animal, plant, and bacterial cells are shown in Fig.

EPSTEIN–BARR VIRUS: 40 YEARS ON - Free download as PDF File .pdf), Text File .txt) or read online for free. nature. Organization of the Epstein-Barr virus DNA molecule.

III. Location of the P3HR-1 deletion junction and characterization of the NotI repeat units that form part of the template for an abundant O-tetradecanoylphorbolacetate-induced mRNA transcr. currently assigned to [{"ult_entity_alias_name"=>"Health and Human Services United States of America As Represented By The SEC", "ult_ent_alias_id"=>, "entity_alias_name"=>"Health and Human Services United States of America As Represented By The SEC", "ent_alias_id"=>, "is_entity"=>true}] asserted in 0 litigation cases est.